Baptista FI

ORCID iD
orcid.org/0000-0001-7972-640X
  • Keywords
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Synapse formation and dysfunction,

Sources:
Baptista FI (2013-11-18)

Cell dysfunction and cell death,

Sources:
Baptista FI (2013-11-18)

Axonal transport,

Sources:
Baptista FI (2013-11-18)

Neurotransmitter release,

Sources:
Baptista FI (2013-11-18)

Exocytosis,

Sources:
Baptista FI (2013-11-18)

Diabetic retinopathy,

Sources:
Baptista FI (2013-11-18)

Neuroinflammation,

Sources:
Baptista FI (2013-11-18)

Diabetic Encephalopathy,

Sources:
Baptista FI (2013-11-18)

Diabetes

Sources:
Baptista FI (2013-11-18)

  • Other IDs
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Scopus Author ID: 22955202000

Sources:
Scopus to ORCID (2014-01-15)

ResearcherID: E-2828-2012

Sources:
Clarivate Analytics (2014-05-16)

Biography

Filipa Baptista graduated in Biology in 2006 from the Faculty of Sciences and Technology of University of Coimbra. During 2007, Filipa participated in several projects that were being developed at the Centre of Ophthalmology and Vision Sciences, IBILI. In 2008, Filipa started her PhD in Biosciences (Faculty of Sciences and Technology, University of Coimbra) under the supervision of Dr. António Francisco Ambrósio. The PhD project was entitled “Impact of diabetes on exocytosis/neurotransmitter release in the retina” (SFRH/BD/35961/2007) and aimed to give further insight into the molecular and cellular alterations in the retina induced by diabetes, giving a particular attention to changes occurring in nerve terminals and to changes occurring in neural cells. In the last year of her PhD, she focused on evaluating the impact of early diabetes in the axonal transport in the retina and also in the hippocampus. Through this research, she became very interested in further studying the impairment of axonal transport in diabetes. Presently, Filipa Baptista is a PostDoc Research fellow in the project “Unravelling the involvement of neuroinflammation on the impairment of axonal transport in hippocampal neurons under diabetic conditions: contribution to synaptic changes and memory impairment” (SFRH/BPD/86830/2012). This project aims to study neuroinflammation-driven changes on axonal transport in the hippocampus of diabetic animals and also evaluate the contribution of neuroinflammation for diabetes-induced memory impairments. Additionally, Filipa is very interested in studying how intrauterine/neonatal diabetic environment affects synapse formation in the offspring of diabetic animals.
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