Inflammatory cytokines in eyes with uveal melanoma and relation with macrophage infiltration.
Applications of the Role of a-MSH in Ocular Immune Privilege.
The immune privileged retina mediates an alternative activation of J774A.1 cells.
Local treatment with alpha-melanocyte stimulating hormone reduces corneal allorejection.
The alpha-melanocyte stimulating hormone induces conversion of effector T cells into treg cells.
Ocular immune privilege in the year 2010: ocular immune privilege and uveitis.
Localized retinal neuropeptide regulation of macrophage and microglial cell functionality.
Injection of an alpha-melanocyte stimulating hormone expression plasmid is effective in suppressing experimental autoimmune uveitis.
Ocular immune privilege.
Review of the activation of TGF-beta in immunity.
Eye-derived cytokines and the immunosuppressive intraocular microenvironment: a review.
Regulation of cytokine-induced human C-reactive protein production by transforming growth factor-beta.
Suppression of nitric oxide generated by inflammatory macrophages by calcitonin gene-related peptide in aqueous humor.
Effect of alpha-2-macroglobulin on cytokine-mediated human C-reactive protein production.
Ocular immunosuppressive microenvironment.
Mice with Th2-biased immune systems accept orthotopic corneal allografts placed in "high risk" eyes.
Characteristics of frozen colostrum thawed in a microwave oven.
Effects of experimental ocular inflammation on ocular immune privilege.
Primary Open-Angle Glaucoma: A Transforming Growth Factor-ß Pathway-Mediated Disease.
Specific aqueous humor factors induce activation of regulatory T cells.
Following EAU recovery there is an associated MC5r-dependent APC induction of regulatory immunity in the spleen.
Neuropeptide regulation of immunity. The immunosuppressive activity of alpha-melanocyte-stimulating hormone (alpha-MSH).
Neural control of ocular immune privilege.
The neuropeptides a-MSH and NPY modulate phagocytosis and phagolysosome activation in RAW 264.7 cells.
Analysis of immunomodulatory activities of aqueous humor from eyes of mice with experimental autoimmune uveitis.
Alpha-melanocyte-stimulating hormone suppresses antigen-stimulated T cell production of gamma-interferon.
Immune response to intragraft antigen in draining lymph nodes after corneal transplantation is mediated by interleukin-12.
Identification of alpha-melanocyte stimulating hormone as a potential immunosuppressive factor in aqueous humor.
IL-6 antagonizes TGF-beta and abolishes immune privilege in eyes with endotoxin-induced uveitis.
The effects of high glucose on human endothelial cell growth and gene expression are not mediated by transforming growth factor-beta.
Immunoreactive vasoactive intestinal peptide contributes to the immunosuppressive activity of normal aqueous humor.
Neuroimmunomodulation and immune privilege: the role of neuropeptides in ocular immunosuppression.
Neuroimmunomodulation in immune privilege: role of neuropeptides in ocular immunosuppression.
Inhibition of antigen-stimulated effector T cells by human cerebrospinal fluid.
Immune deviation in relation to ocular immune privilege.
Immune privilege, T-cell tolerance, and tissue-restricted autoimmunity.
A small molecular weight factor in aqueous humor acts on C1q to prevent antibody-dependent complement activation.
Aqueous humor induces transforming growth factor-beta (TGF-beta)-producing regulatory T-cells.
Inducible immune regulation following autoimmune disease in the immune-privileged eye.
Thrombospondin plays a vital role in the immune privilege of the eye.
Modulation of regulatory T cell immunity by the neuropeptide alpha-melanocyte stimulating hormone.
Somatostatin is an immunosuppressive factor in aqueous humor.
Induction of regulatory T cells by the immunomodulating cytokines alpha-melanocyte-stimulating hormone and transforming growth factor-beta2.
Diminution of experimental autoimmune uveoretinitis (EAU) in mice depleted of NK cells.
Draining lymph nodes of corneal transplant hosts exhibit evidence for donor major histocompatibility complex (MHC) class II-positive dendritic cells derived from MHC class II-negative grafts.
Ocular immune privilege and the impact of intraocular inflammation.
The immunomodulating neuropeptide alpha-melanocyte-stimulating hormone (alpha-MSH) suppresses LPS-stimulated TLR4 with IRAK-M in macrophages.
Melanocortin 5 receptor and ocular immunity.
Alpha-MSH regulates protein ubiquitination in T cells.
Effect of the ocular microenvironment in regulating corneal dendritic cell maturation.
In vitro generated autoimmune regulatory T cells enhance intravitreous allogeneic retinal graft survival.
The diminishment of experimental autoimmune encephalomyelitis (EAE) by neuropeptide alpha-melanocyte stimulating hormone (alpha-MSH) therapy.
Diminishment of alpha-MSH anti-inflammatory activity in MC1r siRNA-transfected RAW264.7 macrophages.
Pigment epithelial growth factor suppresses inflammation by modulating macrophage activation.
An eye's view of T regulatory cells.
Ocular immunosuppressive microenvironment.
Creating an immune-privileged site using retinal progenitor cells and biodegradable polymers.