Peirong Lu received his Ph.D. degree from Kanazawa University at 2004. He did his postdoctoral training at Soochow University. Currently, he is the professor and director of Department of Ophthalmology, the First Affiliated Hospital of Soochow University. His research focuses on ocular neovascularization. He has published more than 20 papers including Invest Ophthalmol & Vis Sci, Journal of Immunology, Cornea, Molecular Vision, and American Journal of Pathology.
1. Liu G, Zhang W, Xiao Y, Lu P*. Critical Role of IP-10 on Reducing Experimental Corneal Neovascularization. Curr Eye Res. 2015 Sep;40(9):891-901. Coorespondence
2. Chen Z, Liu G, Xiao Y, Lu P*. Adrenomedullin22-52 suppresses high-glucose-induced migration, proliferation, and tube formation of human retinal endothelial cells. Mol Vis. 2014 Mar 3;20:259-69. Coorespondence
3. Dai C, Liu G, Li L, Xiao Y, Zhang X, Lu P*. ADP-ribosylation factor as a novel target for corneal neovascularization regression. Mol Vis. 2012;18:2947-2953. Coorespondence
4. Zhou WJ, Liu GQ, Li LB, Zhang XG, Lu PR*. Inhibitory effect of CCR3 signal on alkali-induced corneal neovascularization. Int J Ophthalmol. 2012;5(3):251-257. Coorespondence
5. Lu P*, Li L, Liu G, Baba T, Ishida Y, Nosaka M, Kondo T, Zhang X, Mukaida N. Critical role of TNF-α-induced macrophage VEGF and iNOS production in the experimental corneal neovascularization. Invest Ophthalmol Vis Sci. 2012 Jun 14;53(7):3516-3526. Coorespondence
6. Liu GQ, Lu PR*, Li LB, Zhang XG. Inhibited experimental corneal neovascularization by neutralizing anti-SDF-1α antibody. Int J Ophthalmol. 2012;5(1):7-12. Coorespondence
7. Liu G, Lu P*, Li L, Jin H, He X, Mukaida N, Zhang X. Critical role of SDF-1α-induced progenitor cell recruitment and macrophage VEGF production in the experimental corneal neovascularization. Mol Vis. 2011;17:2129-2138. Coorespondence
8. Chen Y, Liu GQ, Lu PR*. Potential involvement of nitric oxide synthase but not inducible nitric oxide synthase in the development of experimental corneal neovascularization. Int J Ophthalmol. 2011;4(4):343-348. Coorespondence
9. Lu P*, Li L, Liu G, Zhang X, Mukaida N. Enhanced experimental corneal neovascularization along with aberrant angiogenic factor expression in the absence of IL-1 receptor antagonist. Invest Ophthalmol Vis Sci. 2009 Oct;50(10):4761-4768. Coorespondence
10. Lu P*, Li L, Liu G, van Rooijen N, Mukaida N, Zhang X. Opposite roles of CCR2 and CX3CR1 macrophages in alkali-induced corneal neovascularization. Cornea. 2009 Jun;28(5):562-569. Coorespondence
11. Lu P, Li L, Wu Y, Mukaida N, Zhang X. Essential contribution of CCL3 to alkali-induced corneal neovascularization by regulating vascular endothelial growth factor production by macrophages. Mol Vis. 2008 Sep 5;14:1614-1622.
12. Lu P, Li L, Kuno K, Wu Y, Baba T, Li YY, Zhang X, Mukaida N. Protective roles of the fractalkine/CX3CL1-CX3CR1 interactions in alkali-induced corneal neovascularization through enhanced antiangiogenic factor expression. J Immunol. 2008 Mar 15;180(6):4283-4291.
13. Lu P*, Li L, Mukaida N, Zhang X. Alkali-induced corneal neovascularization is independent of CXCR2-mediated neutrophil infiltration. Cornea. 2007 Feb;26(2):199-206. Coorespondence
14. Lu P, Nakamoto Y, Nemoto-Sasaki Y, Fujii C, Wang H, Hashii M, Ohmoto Y, Kaneko S, Kobayashi K, Mukaida N. Potential interaction between CCR1 and its ligand, CCL3, induced by endogenously produced interleukin-1 in human hepatomas. Am J Pathol. 2003 Apr;162(4):1249-1258.