Personal information

Activities

Employment (1)

Yale School of Medicine: New Haven, Connecticut, US

2019-11-01 to present | Postdoctoral Associate (Genetics)
Employment
Source: Self-asserted source
Samuel Hulbert

Education and qualifications (3)

David Geffen School of Medicine at UCLA: Los Angeles, California, US

2020-08-03 to present | MD
Education
Source: Self-asserted source
Samuel Hulbert

Duke University: Durham, NC, US

2013-08-26 to 2019-10-08 | PhD (Neurobiology)
Education
Source: Self-asserted source
Samuel Hulbert

Furman University: Greenville, SC, US

2009-08 to 2013-05 | BS (Neuroscience)
Education
Source: Self-asserted source
Samuel Hulbert

Professional activities (2)

David Geffen School of Medicine at UCLA: Los Angeles, California, US

David Geffen Medical Scholarship
Distinction
Source: Self-asserted source
Samuel Hulbert

Autism Speaks: New York, NY, US

Dennis Weatherstone Predoctoral Fellowship
Distinction
Source: Self-asserted source
Samuel Hulbert

Funding (1)

Investigating Shank3 function during synaptogenesis in mice to define a therapeutic window for ASD.

Award
Autism Speaks (New York, NY, US)
GRANT_NUMBER:

9597

Source: check_circle
Autism Speaks

Works (11)

Effectiveness of Topical ρ-Kinase Inhibitors in Veterans with Severe Glaucoma on Maximally Tolerated Medical Therapy

Optometry and Vision Science
2022-08 | Journal article
Part of ISSN: 1538-9235
Part of ISSN: 1040-5488
Contributors: Reza Kianian; Samuel W. Hulbert; Simon K. Law; JoAnn Giaconi
Source: Self-asserted source
Samuel Hulbert

A Novel Chd8 Mutant Mouse Displays Altered Ultrasonic Vocalizations and Enhanced Motor Coordination

Autism Research
2020-10 | Journal article
Contributors: Samuel W. Hulbert; Xiaoming Wang; Simisola O. Gbadegesin; Qiong Xu; Xiu Xu; Yong‐Hui Jiang
Source: check_circle
Crossref

Postnatal inactivation of Shank3 expression is insufficient to produce behavioral phenotypes associated with germline deficiency

2020-06-01 | Other
Contributors: Samuel William Hulbert; Haidun Yan; Xiaoming Wang; Alexandra L. Bey; Mary S. Lin; Yong-hui Jiang
Source: check_circle
Crossref

Autism-associated CHD8 deficiency impairs axon development and migration of cortical neurons

Molecular Autism
2018 | Journal article
Part of ISSN: 2040-2392
Contributors: Qiong Xu; Yuan-yuan Liu; Xiaoming Wang; Guo-he Tan; Hui-ping Li; Samuel W. Hulbert; Chun-yang Li; Chun-chun Hu; Zhi-qi Xiong; Xiu Xu et al.
Source: Self-asserted source
Samuel Hulbert via Crossref Metadata Search

Environmental enrichment has minimal effects on behavior in the Shank3 complete knockout model of autism spectrum disorder

Brain and Behavior
2018-11 | Journal article
Contributors: Samuel W. Hulbert; Alexandra L. Bey; Yong‐hui Jiang
Source: check_circle
Crossref

Brain region-specific disruption of Shank3 in mice reveals a dissociation for cortical and striatal circuits in autism-related behaviors

Translational Psychiatry
2018-04-27 | Journal article
Contributors: Alexandra L. Bey; Xiaoming Wang; Haidun Yan; Namsoo Kim; Rebecca L. Passman; Yilin Yang; Xinyu Cao; Aaron J. Towers; Samuel W. Hulbert; Lara J. Duffney et al.
Source: check_circle
Crossref

Formation of retinal direction-selective circuitry initiated by starburst amacrine cell homotypic contact

2017-12-18 | Other
Contributors: Thomas A. Ray; Suva Roy; Christopher Kozlowski; Jingjing Wang; Jon Cafaro; Samuel W. Hulbert; Christopher V. Wright; Greg D. Field; Jeremy N. Kay
Source: check_circle
Crossref
grade
Preferred source (of 3)‎

Cellular and Circuitry Bases of Autism: Lessons Learned from the Temporospatial Manipulation of Autism Genes in the Brain

Neuroscience Bulletin
2017-04-02 | Journal article
Source: Self-asserted source
Samuel Hulbert

Monogenic mouse models of autism spectrum disorders: Common mechanisms and missing links

Neuroscience
2016-05 | Journal article
Contributors: S.W. Hulbert; Y.-H. Jiang
Source: check_circle
Crossref

Altered mGluR5-Homer scaffolds and corticostriatal connectivity in a Shank3 complete knockout model of autism

Nature Communications
2016-05-10 | Journal article
Source: Self-asserted source
Samuel Hulbert

Targeting the histone methyltransferase G9a activates imprinted genes and improves survival of a mouse model of Prader–Willi syndrome

Nature Medicine
Journal article
Source: Self-asserted source
Samuel Hulbert