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https://scholar.google.com/citations?view_op=list_works&hl=en&user=1p-Oh8wAAAAJ

Countries

Norway

Activities

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Employment (3)

Norwegian University of Science and Technology: Trondheim, NO

2011-01-04 to present | Researcher (Centre of Molecular Inflammation Research (CEMIR))
Employment
Source: Self-asserted source
Nathalie Niyonzima

National Institutes of Health: Bethesda, MD, US

2017-02-06 to 2018-08-31 | Visiting fellow (NHLBI)
Employment
Source: Self-asserted source
Nathalie Niyonzima

King’s College London: London, GB

2016-03-01 to 2017-02-05 | Visiting fellow (MRC Center for Transplantation)
Employment
Source: Self-asserted source
Nathalie Niyonzima

Education and qualifications (2)

Norwegian University of Science and Technology: Trondheim, NO

2011-01-04 to 2015-04-15 | PhD in Molecular medicine (Department of Cancer Research and Molecular Medicine )
Education
Source: Self-asserted source
Nathalie Niyonzima

Norwegian University of Science and Technology: Trondheim, NO

2008-08-13 to 2010-09-15 | Masters in Molecular medicine (Department of Laboratory medicine, Children’s and Women’s Health )
Education
Source: Self-asserted source
Nathalie Niyonzima

Professional activities (2)

Norsk Kjemisk Selskap: Bergen, Trondheim, NO

2011-01-04 to present
Service
Source: Self-asserted source
Nathalie Niyonzima

International Complement Society : Trondheim, Trondheim, NO

2011-01-04 to present
Service
Source: Self-asserted source
Nathalie Niyonzima

Funding (2)

The role of mitochondrial C5aR1 in driving sterile inflammation in atherosclerosis

2019-01 to 2021-06 | Grant
Centre of Molecular Inflammation Research (CEMIR), Norway (Trondheim, NO)
Source: Self-asserted source
Nathalie Niyonzima

Explore mechanisms by which intracellular complement activation regulates CC-induced inflammasome NLRP3 in monocytes during atherogenesis

2016-01 to 2018-12 | Grant
Norges Forskningsråd (Oslo, Oslo, NO)
GRANT_NUMBER:

251255
Source: Self-asserted source
Nathalie Niyonzima

Works (13)

Mitochondrial C5aR1 activity in macrophages controls IL-1β production underlying sterile inflammation

Science Immunology
2021-12-24 | Journal article
DOI: 10.1126/sciimmunol.abf2489
Contributors: Nathalie Niyonzima; Jubayer Rahman; Natalia Kunz; Erin E. West; Tilo Freiwald; Jigar V. Desai; Nicolas S. Merle; Alexandre Gidon; Bjørnar Sporsheim; Michail S. Lionakis et al.
Source: check_circle
Crossref

SARS-CoV-2 drives JAK1/2-dependent local complement hyperactivation.

Science immunology
2021-04-01 | Journal article
DOI: 10.1126/sciimmunol.abg0833
PMID: 33827897
PMC: PMC8139422
Source: Self-asserted source
Nathalie Niyonzima

Cholesterol crystals use complement to increase NLRP3 signaling pathways in coronary and carotid atherosclerosis.

EBioMedicine
2020-09-11 | Journal article
DOI: 10.1016/j.ebiom.2020.102985
PMID: 32927275
PMC: PMC7494683
Source: Self-asserted source
Nathalie Niyonzima

Complement's favourite organelle-Mitochondria?

British journal of pharmacology
2020-08-25 | Journal article
DOI: 10.1111/bph.15238
PMID: 32840864
Source: Self-asserted source
Nathalie Niyonzima

Complement receptor CD46 co-stimulates optimal human CD8+ T cell effector function via fatty acid metabolism.

Nature communications
2018-10-10 | Journal article
DOI: 10.1038/s41467-018-06706-z
PMID: 30305631
PMC: PMC6180132
Source: Self-asserted source
Nathalie Niyonzima

The intracellular C5 system is critical to DAMP sensing and cellular responses in human monocytes

Regulation and Dysregulation of Innate Immunity in Disease
2018-02-28 | Conference poster
Source: Self-asserted source
Nathalie Niyonzima

The intracellular C5 system is critical to DAMP sensing and cellular responses in human monocytes

2. 16th European meeting on complement in diseases, Copenhagen, Danmark, 2017
2017-09 | Conference abstract
Source: Self-asserted source
Nathalie Niyonzima

Cyclodextrin Reduces Cholesterol Crystal-Induced Inflammation by Modulating Complement Activation.

Journal of immunology (Baltimore, Md. : 1950)
2017-08-30 | Journal article
DOI: 10.4049/jimmunol.1700302
PMID: 28855312
Source: Self-asserted source
Nathalie Niyonzima

Complement activation by cholesterol crystals triggers a subsequent cytokine response.

Molecular immunology
2016-09-28 | Journal article
DOI: 10.1016/j.molimm.2016.09.019
PMID: 27692470
Source: Self-asserted source
Nathalie Niyonzima

Cholesterol Crystals Activate the Lectin Complement Pathway via Ficolin-2 and Mannose-Binding Lectin: Implications for the Progression of Atherosclerosis.

Journal of immunology (Baltimore, Md. : 1950)
2016-05-16 | Journal article
DOI: 10.4049/jimmunol.1502595
PMID: 27183610
Source: Self-asserted source
Nathalie Niyonzima

Reconstituted High-Density Lipoprotein Attenuates Cholesterol Crystal-Induced Inflammatory Responses by Reducing Complement Activation.

Journal of immunology (Baltimore, Md. : 1950)
2015-05-29 | Journal article
DOI: 10.4049/jimmunol.1403044
PMID: 26026058
Source: Self-asserted source
Nathalie Niyonzima

Cholesterol crystal-induced endothelial cell activation is complement-dependent and mediated by TNF.

Immunobiology
2014-07-05 | Journal article
DOI: 10.1016/j.imbio.2014.06.006
PMID: 25053140
Source: Self-asserted source
Nathalie Niyonzima

Cholesterol crystals induce complement-dependent inflammasome activation and cytokine release.

Journal of immunology (Baltimore, Md. : 1950)
2014-02-19 | Journal article
DOI: 10.4049/jimmunol.1302484
PMID: 24554772
PMC: PMC3985066
Source: Self-asserted source
Nathalie Niyonzima
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