Personal information

United States

Activities

Employment (3)

University of Florida: Gainesville, FL, US

2022-09-01 to present | Postdoctoral Associate (Collage of Medicine)
Employment
Source: Self-asserted source
PANKAJ KUMAR SINGH

Lewis Katz School of Medicine at Temple University: Philadelphia, PA, US

2020-10-01 to 2022-08-31 | Postdoctoral fellow (Microbiology and Immunology)
Employment
Source: Self-asserted source
PANKAJ KUMAR SINGH

Institut de Génétique et de Biologie Moléculaire et Cellulaire: Illkirch-Grafenstaden, Grand Est, FR

2013-12 to 2018-06 | Postdoctoral fellow
Employment
Source: Self-asserted source
PANKAJ KUMAR SINGH

Education and qualifications (2)

Indian Institute of Technology Kanpur: Kanpur, Uttar Pradesh, IN

2006-08 to 2013-06 | Graduate student (Biological Sciences and Bioengineering)
Qualification
Source: Self-asserted source
PANKAJ KUMAR SINGH

Banaras Hindu University: Varanasi, Uttar Pradesh, IN

2004-06 to 2006-07 | Master of Science (M.Sc.) (Zoology)
Education
Source: Self-asserted source
PANKAJ KUMAR SINGH

Professional activities (5)

American Society for Cell Biology: Bethesda, MD, US

2021-04-01 to present
Membership
Source: Self-asserted source
PANKAJ KUMAR SINGH

Ataxia UK: London, GB

2017-09 | Young Investigator Bursary
Distinction
Source: Self-asserted source
PANKAJ KUMAR SINGH

National Ataxia Foundation: Minneapolis, Minnesota, US

2017-01 | Post-Doc Fellowship Research Grant
Distinction
Source: Self-asserted source
PANKAJ KUMAR SINGH

Indian National Science Academy: New Delhi, Delhi, IN

2015 | Young scientist medal
Distinction
Source: Self-asserted source
PANKAJ KUMAR SINGH

Department of Science and Technology, Ministry of Science and Technology, India: New Delhi, IN

2011 | International Travel award
Distinction
Source: Self-asserted source
PANKAJ KUMAR SINGH

Funding (1)

Unravelling pathomechanisms of muscle dysfunctionin an autosomal recessive cerebellar ataxia 2 (ARCA2) mice model

Award
National Ataxia Foundation (MN, MN, US)
Part of GRANT_NUMBER: 0
Source: Self-asserted source
PANKAJ KUMAR SINGH

Works (11)

Lysine l-lactylation is the dominant lactylation isomer induced by glycolysis

Nature Chemical Biology
2025-01 | Journal article
Contributors: Di Zhang; Jinjun Gao; Zhijun Zhu; Qianying Mao; Zhiqiang Xu; Pankaj K. Singh; Cornelius C. Rimayi; Carlos Moreno-Yruela; Shuling Xu; Gongyu Li et al.
Source: check_circle
Crossref

Correction to: Interdependence of laforin and malin proteins for their stability and functions could underlie the molecular basis of locus heterogeneity in Lafora disease.

Journal of biosciences
2018-09-01 | Journal article
Source: Self-asserted source
PANKAJ KUMAR SINGH
grade
Preferred source (of 2)‎

Glycogen synthase protects neurons from cytotoxicity of mutant huntingtin by enhancing the autophagy flux.

Cell death & disease
2018-02-08 | Journal article
Source: Self-asserted source
PANKAJ KUMAR SINGH
grade
Preferred source (of 2)‎

Coumarin-pyrene conjugate: Synthesis, structure and Cu-selective fluorescent sensing in mammalian kidney cells

Journal of Luminescence
2016 | Journal article
EID:

2-s2.0-84948750171

Part of ISSN: 00222313
Contributors: Wani, M.A.; Singh, P.K.; Pandey, R.; Pandey, M.D.
Source: Self-asserted source
PANKAJ KUMAR SINGH via Scopus - Elsevier

Cerebellar Ataxia and Coenzyme Q Deficiency through Loss of Unorthodox Kinase Activity.

Molecular cell
2016-08-04 | Journal article
Source: Self-asserted source
PANKAJ KUMAR SINGH
grade
Preferred source (of 2)‎

Changing shapes of glycogen-autophagy nexus in neurons: perspective from a rare epilepsy.

Frontiers in neurology
2015-02-04 | Journal article
Source: Self-asserted source
PANKAJ KUMAR SINGH
grade
Preferred source (of 2)‎

Decreased O-linked GlcNAcylation protects from cytotoxicity mediated by huntingtin exon1 protein fragment.

The Journal of biological chemistry
2014-03-19 | Journal article
Source: Self-asserted source
PANKAJ KUMAR SINGH
grade
Preferred source (of 2)‎

Activation of serum/glucocorticoid-induced kinase 1 (SGK1) underlies increased glycogen levels, mTOR activation, and autophagy defects in Lafora disease.

Molecular biology of the cell
2013-10-16 | Journal article
Source: Self-asserted source
PANKAJ KUMAR SINGH
grade
Preferred source (of 2)‎

Lafora disease E3 ubiquitin ligase malin is recruited to the processing bodies and regulates the microRNA-mediated gene silencing process via the decapping enzyme Dcp1a.

RNA biology
2012-11-06 | Journal article
Source: Self-asserted source
PANKAJ KUMAR SINGH
grade
Preferred source (of 2)‎

The laforin-malin complex negatively regulates glycogen synthesis by modulating cellular glucose uptake via glucose transporters.

Molecular and cellular biology
2011-11-28 | Journal article
Source: Self-asserted source
PANKAJ KUMAR SINGH
grade
Preferred source (of 2)‎

The malin-laforin complex suppresses the cellular toxicity of misfolded proteins by promoting their degradation through the ubiquitin-proteasome system.

Human molecular genetics
2008-11-25 | Journal article
Source: Self-asserted source
PANKAJ KUMAR SINGH
grade
Preferred source (of 2)‎

Peer review (4 reviews for 4 publications/grants)

Review activity for Heliyon. (1)
Review activity for Journal of clinical neuroscience. (1)
Review activity for Journal of Genetics. (1)
Review activity for Molecular neurobiology. (1)