Manuel Blandino-Rosano (0000-0002-1712-8233)

Personal information

Biography

Birth Date: June 3, 1978
Birthplace/Citizenship: Cádiz/Spain
Address: University of Miami. Department of Internal Medicine, Division of Endocrinology, Diabetes and Metabolism. 1580 NW 10th Avenue. Batchelor Bldg 6th Floor. Miami, Florida, 33136
Email: mblandino@med.miami.edu

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University of Miami: Miami, FL, US

2015-09-01 to present | Research Assistant Professor (Medicine)

Employment

Manuel Blandino-Rosano

https://orcid.org/0000-0002-1712-8233

Personal information

Other IDs

Biography

Activities

Employment (4)

University of Miami: Miami, FL, US

University of Michigan: Ann Arbor, MI, US

University of Michigan: Ann Arbor, MI, US

Universidad de Cádiz: Cadiz, Andalucía, ES

Professional activities (1)

Sociedad Española de Diabetes: Madrid, ES

Works (40)

Loss of O-GlcNAcylation modulates mTORC1 and autophagy in β cells, driving diabetes 2 progression

Trimethylguanosine synthase 1 is a novel regulator of pancreatic beta-cell mass and function

Novel roles of mTORC2 in regulation of insulin secretion by actin filament remodeling

Glucagon Resistance and Decreased Susceptibility to Diabetes in a Model of Chronic Hyperglucagonemia

TGS1: a novel regulator of $β$-cell mass and function

The nutrient sensor mTORC1 regulates insulin secretion by modulating ß-cell autophagy

2124-P: Raptor Levels Are Critical in the Adaptation of Beta Cells to High-Fat Diet

Glucose stimulates microRNA-199 expression in murine pancreatic $β$-cells

Glucose stimulates microRNA-199 expression in murine pancreatic β-cells

Glucose stimulates microRNA-199 expression in murine pancreatic β-cells

GLP-1 mediated improvement of the glucose tolerance in the T2DM GK rat model after massive jejunal resection

Steroid-Free Immune Suppression Impairs Glycemic Control in a Healthy Cynomolgus Monkey

Androgen excess in pancreatic $β$ cells and neurons predisposes female mice to type 2 diabetes

Androgen excess in pancreatic β cells and neurons predisposes female mice to type 2 diabetes

Bariatric surgery influences $β$-cell turnover in non obese rats.

Loss of mTORC1 signaling alters pancreatic $α$ cell mass and impairs glucagon secretion

Loss of mTORC1 signalling impairs $β$-cell homeostasis and insulin processing

Overexpression of kinase-dead mTOR impairs glucose homeostasis by regulating insulin secretion and not $β$-cell mass

Role of nutrients and mTOR signaling in the regulation of pancreatic progenitors development

Loss of mTORC1 signaling alters pancreatic α cell mass and impairs glucagon secretion

4E-BP2/SH2B1/IRS2 are part of a novel feedback loop that controls $β$-cell mass

Genetic Dissection of the mTORC1 Pathway Reveals a Link between Nutrient Signals and Regulation of Hormone Processing and Beta-Cell Mass

Natural history of $β$-cell adaptation and failure in type 2 diabetes

Beta-Cell Raptor Deletion Results in Reduced Beta-Cell Mass, Diabetes, and Impaired Adaptation to Insulin Resistance by Enhanced Authophagy

Exposure of mouse embryonic pancreas to metformin enhances the number of pancreatic progenitors

Long-lived crowded-litter mice exhibit lasting effects on insulin sensitivity and energy homeostasis

Nitric oxide is a mediator of antiproliferative effects induced by proinflammatory cytokines on pancreatic beta cells

Enhanced beta cell proliferation in mice overexpressing a constitutively active form of Akt and one allele of p21 Cip

mTORC1 signaling and regulation of pancreatic $β$-cell mass

Decrease in $β$-cell proliferation precedes apoptosis during diabetes development in bio-breeding/worcester rat: beneficial role of exendin-4

Decrease in ß-Cell Proliferation Precedes Apoptosis during Diabetes Development in Bio-Breeding/Worcester Rat: Beneficial Role of Exendin-4

Decreased IRS signaling impairs $β$-cell cycle progression and survival in transgenic mice overexpressing S6K in $β$-cells

Effects of ghrelin treatment to promote proliferation of pancreatic islet in BB rats

TSC1/TSC2 Signaling in Pancreatic $β$-Cells

Anti-proliferative effect of pro-inflammatory cytokines in cultured b cells is associated with extracellular signal-regulated kinase 1/2 pathway inhibition: protective role of glucagon-like peptide-1

Anti-proliferative Effect of Pro-inflammatory Cytokines in Cultured Beta Cells is Associated with Erk1/2 Pathway Inhibition: Protective Role of GLP-1 2

Effects of exendin-4 and interferon-gamma antibody on beta cell apoptosis and proliferation in BB rat

Casado-Medrano, Victoria, 1300 Cerione, Richard A., 1328 Chung, Jihyun, 1165 Clarke, Andrew M., 1288

Diabetes Volume: 59 ISSN: 1939-327X ISO Abbreviation: Diabetes Publication Date: 2010 Oct

From Cell Biology to Tissue Engineering