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Scopus Author ID: 57211476765

Biography

Tomonori Masuda
Affiliation: Department of Gastroenterology and Hepatology, Kyoto University Graduate School of Medicine
Research Field: Pancreatic Ductal Adenocarcinoma

Education & Professional Experience:
Education:

Faculty of Medicine, Kyoto University (April 1, 2004 - March 10, 2011)
Department of Gastroenterology and Hepatology, Kyoto University Graduate School of Medicine (April 1, 2018 - March 23, 2022)

Professional Experience:
Resident Physician, Kyoto University Hospital (April 2011 - March 2013)
Gastroenterologist, Japanese Red Cross Wakayama Medical Center (April 2013 - March 2018)
Gastroenterologist, Kyoto University Hospital (April 2018 - Present)

Research Achievements & Specialized Knowledge:
Tomonori Masuda specializes in Pancreatic Ductal Adenocarcinoma with a particular focus on analyzing pancreatic cancer development and metastasis. His expertise includes investigating the role of RECK, as demonstrated in the study by Masuda, T. et al. (2023) titled "Pancreatic RECK inactivation promotes cancer formation, epithelial-mesenchymal transition, and metastasis" published in the Journal of Clinical Investigation.

Other Activities:
September 23, 2023: Featured in Asahi Shimbun and other media outlets for research on pancreatic cancer. "Pancreatic Cancer Progression: Reduction of Specific Protein RECK Increases Susceptibility to Metastasis, Revealed by Kyoto University"

Activities

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Employment (1)

Kyoto University Hospital: Kyoto, 54 Shogoin-kawahara-cho, Sakyo-ku, JP

2022-04-01 to present | Clinical Fellow (Gastroenterology and Hepatology)
Employment
Source: Self-asserted source
Tomonori Masuda

Education and qualifications (1)

Kyoto University: Kyoto, JP

(Gastroenterology and hepatology)
Education
Source: Self-asserted source
Tomonori Masuda

Works (10)

THBS1-producing tumor-infiltrating monocyte-like cells contribute to immunosuppression and metastasis in colorectal cancer.

Nature communications
2023-09-25 | Journal article | Author
DOI: 10.1038/s41467-023-41095-y
PMID: 37749092
PMC: PMC10520015
Contributors: Omatsu M; Yuki Nakanishi; Iwane K; Aoyama N; Duran A; Yu Muta; Anxo Martinez-Ordoñez; Han Q; Agatsuma N; Mizukoshi K et al.
Source: Self-asserted source
Tomonori Masuda

Pancreatic RECK inactivation promotes cancer formation, epithelial-mesenchymal transition, and metastasis

Journal of Clinical Investigation
2023-09-15 | Journal article
DOI: 10.1172/JCI161847
Contributors: Tomonori Masuda; Akihisa Fukuda; Go Yamakawa; Mayuki Omatsu; Mio Namikawa; Makoto Sono; Yuichi Fukunaga; Munemasa Nagao; Osamu Araki; Takaaki Yoshikawa et al.
Source: check_circle
Crossref

Brg1 controls stemness and metastasis of pancreatic cancer through regulating hypoxia pathway

Oncogene
2023-06-23 | Journal article
DOI: 10.1038/s41388-023-02716-4
Contributors: Osamu Araki; Motoyuki Tsuda; Mayuki Omatsu; Mio Namikawa; Makoto Sono; Yuichi Fukunaga; Tomonori Masuda; Takaaki Yoshikawa; Munemasa Nagao; Satoshi Ogawa et al.
Source: check_circle
Crossref

Simultaneous activation of Kras-Akt and Notch pathways induces extrahepatic biliary cancer via the mTORC1 pathway.

The Journal of pathology
2023-06-13 | Journal article
DOI: 10.1002/path.6139
PMID: 37310065
Contributors: Mio Namikawa; Fukuda A; Mizukoshi K; Iwane K; Kawai M; Yamakawa G; Omatsu M; Sono M; Masuda T; Araki O et al.
Source: Self-asserted source
Tomonori Masuda

JNK pathway plays a critical role for expansion of human colorectal cancer in the context of BRG1 suppression.

Cancer science
2022-08-17 | Journal article
DOI: 10.1111/cas.15520
PMID: 35924439
PMC: PMC9530857
Contributors: Takaaki Yoshikawa; Fukuda A; Omatsu M; Namikawa M; Sono M; Fukunaga Y; Masuda T; Araki O; Munemasa Nagao; Ogawa S et al.
Source: Self-asserted source
Tomonori Masuda

Concurrent Activation of Kras and Canonical Wnt Signaling Induces Premalignant Lesions That Progress to Extrahepatic Biliary Cancer in Mice

Cancer Research
2022-05-03 | Journal article
DOI: 10.1158/0008-5472.CAN-21-2176
Contributors: Munemasa Nagao; Akihisa Fukuda; Mayuki Omatsu; Mio Namikawa; Makoto Sono; Yuichi Fukunaga; Tomonori Masuda; Osamu Araki; Takaaki Yoshikawa; Satoshi Ogawa et al.
Source: check_circle
Crossref
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Preferred source (of 10)‎

Loss of Arid1a and Pten in Pancreatic Ductal Cells Induces Intraductal Tubulopapillary Neoplasm via the YAP/TAZ Pathway.

Gastroenterology
2022-04-25 | Journal article
DOI: 10.1053/j.gastro.2022.04.020
PMID: 35483445
Contributors: Fukunaga Y; Fukuda A; Omatsu M; Namikawa M; Sono M; Masuda T; Araki O; Munemasa Nagao; Yoshikawa T; Ogawa S et al.
Source: Self-asserted source
Tomonori Masuda

Brg1 is required to maintain colorectal cancer stem cells.

The Journal of pathology
2021-08-20 | Journal article
DOI: 10.1002/path.5759
PMID: 34415580
Contributors: Takaaki Yoshikawa; Akihisa Fukuda; Omatsu M; Namikawa M; Sono M; Fukunaga Y; Masuda T; Araki O; Munemasa Nagao; Ogawa S et al.
Source: Self-asserted source
Tomonori Masuda

SETDB1 Inhibits p53-Mediated Apoptosis and Is Required for Formation of Pancreatic Ductal Adenocarcinomas in Mice.

Gastroenterology
2020-04-28 | Journal article
DOI: 10.1053/j.gastro.2020.04.047
PMID: 32360551
Contributors: Ogawa S; Fukuda A; Matsumoto Y; Hanyu Y; Sono M; Fukunaga Y; Masuda T; Araki O; Munemasa Nagao; Takaaki Yoshikawa et al.
Source: Self-asserted source
Tomonori Masuda

Epithelial EP4 plays an essential role in maintaining homeostasis in colon.

Scientific reports
2019-10-23 | Journal article
DOI: 10.1038/s41598-019-51639-2
PMID: 31645712
PMC: PMC6811535
Contributors: Matsumoto Y; Nakanishi Y; Yoshioka T; Yamaga Y; Masuda T; Fukunaga Y; Sono M; Takaaki Yoshikawa; Munemasa Nagao; Araki O et al.
Source: Self-asserted source
Tomonori Masuda
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