Personal information

United States

Activities

Employment (1)

Johns Hopkins Bloomberg School of Public Health: Md., Md., US

2013-12-01 to present | Research Associate (Molecular Microbiology and Immunology)
Employment
Source: Self-asserted source
Heather Lamb

Education and qualifications (1)

Washington University in St. Louis: MO, MO, US

2001-08-01 to 2008-08-15 | Ph.D. (Cell Biology and Physiology)
Education
Source: Self-asserted source
Heather Lamb

Works (18)

Double agents of cell death: novel emerging functions of apoptotic regulators.

The FEBS journal
2020-04-11 | Journal article
Source: Self-asserted source
Heather Lamb

The Dark Side of Estrogen Stops Translation to Induce Apoptosis.

Molecular cell
2019-09-01 | Journal article
Source: Self-asserted source
Heather Lamb

Overview of BCL-2 Family Proteins and Therapeutic Potentials.

Methods in molecular biology (Clifton, N.J.)
2019-01-01 | Journal article
Source: Self-asserted source
Heather Lamb

KCTD7 deficiency defines a distinct neurodegenerative disorder with a conserved autophagy-lysosome defect.

Annals of neurology
2018-11-08 | Journal article
Source: Self-asserted source
Heather Lamb

Connecting mitochondrial dynamics and life-or-death events via Bcl-2 family proteins.

Neurochemistry international
2017-04-28 | Journal article
Source: Self-asserted source
Heather Lamb

Unlatched BAX pairs for death.

Cell
2013-01-01 | Journal article
Source: Self-asserted source
Heather Lamb

A highly toxic cellular prion protein induces a novel, nonapoptotic form of neuronal death.

The American journal of pathology
2010-05-14 | Journal article
Source: Self-asserted source
Heather Lamb

Optimal promoter usage for lentiviral vector-mediated transduction of cultured central nervous system cells.

Journal of neuroscience methods
2010-03-27 | Journal article
Source: Self-asserted source
Heather Lamb

Noncanonical functions of BCL-2 proteins in the nervous system.

Advances in experimental medicine and biology
2010-01-01 | Journal article
Source: Self-asserted source
Heather Lamb

A deleted prion protein that is neurotoxic in vivo is localized normally in cultured cells.

Journal of neurochemistry
2008-11-12 | Journal article
Source: Self-asserted source
Heather Lamb

Prion protein lacks robust cytoprotective activity in cultured cells.

Molecular neurodegeneration
2008-08-21 | Journal article
Source: Self-asserted source
Heather Lamb

Non-infectious aggregates of the prion protein react with several PrPSc-directed antibodies.

Journal of neurochemistry
2008-06-01 | Journal article
Source: Self-asserted source
Heather Lamb

The cellular prion protein (PrP(C)): its physiological function and role in disease.

Biochimica et biophysica acta
2007-03-02 | Journal article
Source: Self-asserted source
Heather Lamb

Neonatal lethality in transgenic mice expressing prion protein with a deletion of residues 105-125.

The EMBO journal
2007-01-01 | Journal article
Source: Self-asserted source
Heather Lamb

Histone deacetylase-dependent establishment and maintenance of broad low-level histone acetylation within a tissue-specific chromatin domain.

Biochemistry
2002-12-01 | Journal article
Source: Self-asserted source
Heather Lamb

Distinct mechanisms control RNA polymerase II recruitment to a tissue-specific locus control region and a downstream promoter.

Molecular cell
2001-08-01 | Journal article
Source: Self-asserted source
Heather Lamb

Developmentally dynamic histone acetylation pattern of a tissue-specific chromatin domain.

Proceedings of the National Academy of Sciences of the United States of America
2000-12-01 | Journal article
Source: Self-asserted source
Heather Lamb

Linking Notch signaling, chromatin remodeling, and T-cell leukemogenesis.

Journal of cellular biochemistry. Supplement
2000-01-01 | Journal article
Source: Self-asserted source
Heather Lamb

Peer review (4 reviews for 4 publications/grants)

Review activity for Cell communication and signaling. (1)
Review activity for Cell death and differentiation. (1)
Review activity for Journal of translational medicine. (1)
Review activity for PloS one. (1)