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Employment (1)

Mossakowski Medical Research Institute: Warsaw, PL

(Department of Neurotoxicology)
Employment
Source: Self-asserted source
Monika Szeliga

Funding (4)

Role of GAB glutaminase in glioblastoma.

2022-01 to 2025-12 | Grant
National Science Centre Poland (Cracow, PL)
Source: Self-asserted source
Monika Szeliga

Peroxiredoxins as potential therapeutic targets in glioblastoma.

2018-01 to 2023-01 | Grant
National Science Centre (Cracow, PL)
GRANT_NUMBER:

2017/25/B/NZ7/00388

Source: Self-asserted source
Monika Szeliga

New 1,3,4-thiadiazole derivatives as inhibitors of critical steps of the glutamine-glutamate cycle in glioblastoma cells.

2014 to 2017 | Grant
National Science Centre (Cracow, PL)
GRANT_NUMBER:

2013/11/D/NZ7/00925

Source: Self-asserted source
Monika Szeliga

The influence of the modulation of glutaminase expression on the phenotype of glioblastoma cells and their sensitivity to alkylating agents.

2010 to 2013 | Grant
Ministry of Science and Higher Education (Warsaw, PL)
GRANT_NUMBER:

No N N401 039238

Source: Self-asserted source
Monika Szeliga

Works (33)

Disrupted glutamate homeostasis as a target for glioma therapy

Pharmacological Reports
2024-12 | Journal article
Contributors: Mikołaj Biegański; Monika Szeliga
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Crossref

Two Faces of Glutaminase GLS2 in Carcinogenesis

Cancers
2023-11-24 | Journal article
Contributors: Joanna Buczkowska; Monika Szeliga
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Crossref
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Preferred source (of 2)‎

Conoidin A, a Covalent Inhibitor of Peroxiredoxin 2, Reduces Growth of Glioblastoma Cells by Triggering ROS Production

Cells
2023-07-26 | Journal article
Contributors: Monika Szeliga; Radosław Rola
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Crossref
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Menadione Potentiates Auranofin-Induced Glioblastoma Cell Death

International Journal of Molecular Sciences
2022-12-11 | Journal article
Contributors: Monika Szeliga; Radosław Rola
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Crossref
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Design, Synthesis and Activity of New N1-Alkyl Tryptophan Functionalized Dendrimeric Peptides against Glioblastoma

Biomolecules
2022-08-13 | Journal article
Contributors: Marta Sowińska; Monika Szeliga; Maja Morawiak; Barbara Zabłocka; Zofia Urbanczyk-Lipkowska
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Crossref
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Comprehensive analysis of the expression levels and prognostic values of PRDX family genes in glioma

Neurochemistry International
2022-02 | Journal article
Contributors: Monika Szeliga
Source: check_circle
Crossref

Roles of nitric oxide and polyamines in brain tumor growth

Advances in Medical Sciences
2021-03 | Journal article
Contributors: Monika Szeliga; Jan Albrecht
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Crossref

Peptide Dendrimers with Non-Symmetric Bola Structure Exert Long Term Effect on Glioblastoma and Neuroblastoma Cell Lines

Biomolecules
2021-03-15 | Journal article
Contributors: Marta Sowińska; Monika Szeliga; Maja Morawiak; Elżbieta Ziemińska; Barbara Zabłocka; Zofia Urbańczyk-Lipkowska
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Crossref
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Preferred source (of 2)‎

Design, synthesis and biological evaluation of novel 1,3,4-thiadiazole derivatives as anti-glioblastoma agents targeting the AKT pathway

Bioorg Chem
2020-12 | Journal article
Source: Self-asserted source
Monika Szeliga

Peroxiredoxins in Neurodegenerative Diseases

Antioxidants
2020-11-30 | Journal article
Contributors: Monika Szeliga
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Crossref
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Thiadiazole derivatives as anticancer agents

Pharmacological Reports
2020-10-03 | Journal article
Contributors: Monika Szeliga
Source: check_circle
Crossref

Erratum: Majewska, E. et al. Transfection with GLS2 Glutaminase (GAB) Sensitizes Human Glioblastoma Cell Lines to Oxidative Stress by a Common Mechanism Involving Suppression of the PI3K/AKT Pathway. Cancers 2019, 11, 115

Cancers
2020-07-27 | Journal article
Contributors: Ewelina Majewska; Javier Márquez; Jan Albrecht; Monika Szeliga
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Crossref
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Preferred source (of 2)‎

Targeting Glutamine Addiction in Gliomas

Cancers
2020-01 | Journal article | Author
Contributors: Marta Obara-Michlewska; Monika Szeliga
Source: check_circle
Multidisciplinary Digital Publishing Institute
grade
Preferred source (of 2)‎

Transfection with GLS2 Glutaminase (GAB) Sensitizes Human Glioblastoma Cell Lines to Oxidative Stress by a Common Mechanism Involving Suppression of the PI3K/AKT Pathway

Cancers
2019-01 | Journal article | Author
Contributors: Ewelina Majewska; FRANCISCO JAVIER MARQUEZ GOMEZ; Jan Albrecht; Monika Szeliga
Source: check_circle
Multidisciplinary Digital Publishing Institute
grade
Preferred source (of 2)‎

Transcription factor GATA3 expression is induced by GLS2 overexpression in a glioblastoma cell line but is GLS2-independent in patient-derived glioblastoma.

J Physiol Pharmacol.
2017-04 | Journal article
Source: Self-asserted source
Monika Szeliga

AKT/GSK3β Signaling in Glioblastoma.

Neurochem Res.
2017-03 | Journal article
Source: Self-asserted source
Monika Szeliga

Expression of prophage-encoded endolysins contributes to autolysis of Lactococcus lactis.

Appl Microbiol Biotechnol.
2017-02 | Journal article
Source: Self-asserted source
Monika Szeliga

Glutamine Metabolism in Gliomas.

Adv Neurobiol.
2016 | Journal article
Source: Self-asserted source
Monika Szeliga

Downregulation of GLS2 in glioblastoma cells is related to DNA hypermethylation but not to the p53 status.

Mol Carcinog.
2016-09 | Journal article
Source: Self-asserted source
Monika Szeliga

Expression of RNAs Coding for Metal Transporters in Blood of Patients with Huntington's Disease.

Neurochem Res.
2016-02 | Journal article
Source: Self-asserted source
Monika Szeliga

Glutaminases in slowly proliferating gastroenteropancreatic neuroendocrine neoplasms/tumors (GEP-NETs): Selective overexpression of mRNA coding for the KGA isoform.

Exp Mol Pathol.
2016-02 | Journal article
Source: Self-asserted source
Monika Szeliga

Opposing roles of glutaminase isoforms in determining glioblastoma cell phenotype.

Neurochem Int.
2015-09 | Journal article
Source: Self-asserted source
Monika Szeliga

Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells.

J Mol Med (Berl).
2014-03 | Journal article
Source: Self-asserted source
Monika Szeliga

Silencing of GLS and overexpression of GLS2 genes cooperate in decreasing the proliferation and viability of glioblastoma cells.

Tumour Biol.
2014-03 | Journal article
Source: Self-asserted source
Monika Szeliga

AcmD, a homolog of the major autolysin AcmA of Lactococcus lactis, binds to the cell wall and contributes to cell separation and autolysis.

PLoS One.
2013-08 | Journal article
Source: Self-asserted source
Monika Szeliga

Transfection of a human glioblastoma cell line with liver-type glutaminase (LGA) down-regulates the expression of DNA-repair gene MGMT and sensitizes the cells to alkylating agents.

J Neurochem.
2012-11 | Journal article
Source: Self-asserted source
Monika Szeliga

2-Amino-1,3,4-thiadiazole derivative (FABT) inhibits the extracellular signal-regulated kinase pathway and induces cell cycle arrest in human non-small lung carcinoma cells.

Bioorg Med Chem Lett.
2012-09 | Journal article
Source: Self-asserted source
Monika Szeliga

Down-regulation of Kir4.1 in the cerebral cortex of rats with liver failure and in cultured astrocytes treated with glutamine: Implications for astrocytic dysfunction in hepatic encephalopathy.

J Neurosci Res.
2011-12 | Journal article
Source: Self-asserted source
Monika Szeliga

Glutamine in neoplastic cells: focus on the expression and roles of glutaminases.

Neurochem Int.
2009-07 | Journal article
Source: Self-asserted source
Monika Szeliga

Transfection with liver-type glutaminase cDNA alters gene expression and reduces survival, migration and proliferation of T98G glioma cells.

Glia
2009-07 | Journal article
Source: Self-asserted source
Monika Szeliga

Regulation of pH in the mammalian central nervous system under normal and pathological conditions: facts and hypotheses.

Neurochem Int.
2008-05 | Journal article
Source: Self-asserted source
Monika Szeliga

Relative expression of mRNAS coding for glutaminase isoforms in CNS tissues and CNS tumors.

Neurochem Res.
2008-05 | Journal article
Source: Self-asserted source
Monika Szeliga

Lack of expression of the liver-type glutaminase (LGA) mRNA in human malignant gliomas.

Neurosci Lett.
2005-02 | Journal article
Source: Self-asserted source
Monika Szeliga

Peer review (16 reviews for 8 publications/grants)

Review activity for Biomedicines. (1)
Review activity for Cellular and molecular neurobiology. (2)
Review activity for Diseases (1)
Review activity for Journal of biomedical science (1)
Review activity for Journal of clinical medicine. (1)
Review activity for Pharmaceuticals. (2)
Review activity for Pharmaceutics. (1)
Review activity for Pharmacological Reports. (7)